Supports Wolfram Mathematica 12.x Permalink. Im having issues with my Softmax Classifier.Invasive assessment of coronary microvascular dysfunction in hypertrophic cardiomyopathy: the index of microvascular resistanceDeriving the softmax function for multinomial (multi-class) classification problems starting from simple logistic regression Using the softmax activation function in the output layer of a deep neural net to represent a categorical distribution over class labels, and obtaining the probabilities of each input element belonging to a labelProjects. Im following the Stanford Machine Learning course while simultaneously developing my Mathematica abilities. If you want to predict the number of dollars (the price) at which a house will be sold. Regression is the hammer we reach for when we want to answer how much or how many questions. In Section 3.1 we introduced linear regression, and worked through building everything from scratch in Section 3.2 and using Gluon in Section 3.3 to automate the most repetitive work.

This is the first such case that documents this abnormality invasively using the IMR. Pressure wire measurements of fractional flow reserve (FFR), coronary flow reserve (CFR) and index of microvascular resistance (IMR) demonstrated a significant microcirculatory dysfunction. Coronary angiography revealed normal epicardial arteries. It’s for Mathematica onlyInternational Nuclear Information System (INIS)GutiÃ©rrez-Barrios, Alejandro Camacho-Jurado, Francisco DÃaz-Retamino, Enrique Gamaza-ChuliÃ¡n, Sergio Agarrado-Luna, Antonio Oneto-Otero, JesÃºs Del Rio-Lechuga, Ana Benezet-Mazuecos, JavierSummary: We present a review of microvascular dysfunction in hypertrophic cardiomyopathy (HCM) and an interesting case of a symptomatic familial HCM patient with inducible ischemia by single photon emission computed tomography. Does not work for other Wolfram products.

The effects of hyperthyroidism on the microcirculation are still controversial in experimental models. This index may be useful in future researches of HCM.Energy Technology Data Exchange (ETDEWEB)GutiÃ©rrez-Barrios, Alejandro, E-mail: [Cardiology Department, Jerez Hospital, Jerez (Spain) Camacho-Jurado, Francisco [Cardiology Department, Punta Europa Hospital, Algeciras (Spain) DÃaz-Retamino, Enrique Gamaza-ChuliÃ¡n, Sergio Agarrado-Luna, Antonio Oneto-Otero, JesÃºs Del Rio-Lechuga, Ana Benezet-Mazuecos, Javier [Cardiology Department, Jerez Hospital, Jerez (Spain)Cardiac microvascular rarefaction in hyperthyroidism- induced left ventricle dysfunction.Freitas, Felipe Estato, Vanessa Carvalho, VinÃcius Frias Torres, Rafael Carvalho Lessa, Marcos Adriano TibiriÃ§Ã¡, EduardoThe pathophysiology underlying hyperthyroidism- induced left ventricle (LV) dysfunction and hypertrophy directly involves the heart and indirectly involves the neuroendocrine systems. We report a case in which microvascular dysfunction is assessed by IMR. Â€¢ However assessment of IMR in HCM has not been previously reported. Â€¢ The index of microvascular resistance (IMR) is a new method for invasively assessing the state of the coronary microcirculation using a single pressure-temperature sensor-tipped coronary wire. - Highlights: â€¢ Microvascular dysfunction is a common feature in hypertrophic cardiomyopathy (HCM) and represents a strong predictor of unfavorable outcome and cardiovascular mortality.

Hyperthyroidism was found to induce profound cardiovascular alterations, such as systolic hypertension, tachycardia, LV dysfunction, cardiac hypertrophy, and myocardial fibrosis. The structural capillary density and the expression of angiotensin II AT1 receptor in the LV were analyzed using histochemistry and immunohistochemistry, respectively. An echocardiographic study was performed to evaluate the alterations in cardiac function, structure and geometry. Male Wistar rats (170-250 g) were divided into two groups: the euthyroid group (n = 10), which was treated with 0.9% saline solution, and the hyperthyroid group (n = 10), which was treated with l-thyroxine (600 Î¼g/kg/day, i.p.) during 14 days.

Â€¢ MEG3 regulates endothelial cell function through PI3k/Akt signaling.Qiu, Gui-Zhen Tian, Wei Fu, Hai-Tao Li, Chao-Peng Liu, BanMicrovascular dysfunction is an important characteristic of diabetic retinopathy. Â€¢ MEG3 regulates retinal endothelial cell function inÂ vitro. Â€¢ MEG3 knockdown aggravates retinal vascular dysfunction inÂ vivo.

In this study, we investigated the role of lncRNA-MEG3 in diabetes-related microvascular dysfunction.

Mathematica 11.2 Problem Softmax